Urinary System

Kidney

Gross Anatomy · Nephron · Filtration Barrier · JG Complex · Embryology · Clinical
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Gross Anatomy

Paired retroperitoneal organs (~10-12 cm, 150 g). Right kidney slightly lower (liver). T12-L3 vertebral levels.

External Anatomy

  • Renal capsule — fibrous outer covering
  • Gerota fascia — perirenal fat cushions kidney
  • Hilum (medial) — contains Renal Vein (anterior), Artery (middle), Ureter (posterior) — VAU mnemonic

Internal Anatomy

  • Cortex — outer; glomeruli, PCT, DCT, cortical collecting ducts
  • Medulla — inner; loops of Henle, vasa recta, medullary collecting ducts; renal pyramids → papillae → minor calyces → major calyces → renal pelvis → ureter
VesselNotes
Renal artery → segmental → interlobar → arcuate → interlobularEnd arteries; no anastomoses → infarcts are wedge-shaped
Afferent → glomerulus → efferent arterioleUnique portal-like arrangement
Vasa rectaLong loops into medulla; countercurrent exchange; concentrate urine
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Nephron

~1 million nephrons per kidney. Cortical nephrons (85%) = short loops. Juxtamedullary (15%) = long loops; key for concentration.

SegmentKey FunctionReabsorbs
PCTBulk iso-osmotic reabsorption67% Na, H2O; ALL glucose, AA, HCO3
Thin descending loopH2O permeable only (concentrates)H2O passively
Thick ascending loopNKCC2; H2O impermeable; dilutes fluidNa, K, Cl (furosemide target)
DCTCa2+ (PTH); NCC (thiazide target)Na, Cl, Ca2+
Collecting ductAldosterone → Na/K; ADH → H2O (AQP2)Na (aldosterone); H2O (ADH)
🧠 Mnemonic

Diuretic targets: Loop (furosemide) → NKCC2 in thick ascending limb. Thiazide → NCC in DCT. Amiloride/triamterene → ENaC in collecting duct. Acetazolamide → carbonic anhydrase in PCT.

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Glomerular Filtration Barrier

Three layers filter ~180 L/day by size AND charge:

  • Fenestrated endothelium — 70-100 nm pores; bars cells; negatively charged glycocalyx
  • GBM — type IV collagen + heparan sulfate (negative charge repels albumin)
  • Podocytes — foot processes + nephrin-bridged filtration slits; main size barrier
🩺 Clinical

Loss of GBM charge (minimal change disease) → albumin leaks → nephrotic syndrome (proteinuria >3.5 g/day, hypoalbuminemia, edema, hyperlipidemia). GFR normal (no GBM destruction). Responds to steroids.

GFR ~125 mL/min normal. Estimated by CKD-EPI using creatinine. Inulin clearance = gold standard. Cystatin C more accurate in some populations.

Juxtaglomerular Complex

  • JG cells (granular cells) — modified smooth muscle in afferent arteriole; secrete renin in response to: ↓BP, ↑sympathetics, ↓NaCl at macula densa
  • Macula densa — specialized DCT cells sensing NaCl; ↓NaCl → signal JG cells to release renin
  • Lacis cells — between macula densa and JG cells; signaling role

RAAS

Renin → Angiotensinogen → Ang I → (ACE in lung) → Ang II → ↑Aldosterone + ↑vasoconstriction + ↑ADH + ↑thirst

⭐ Boards Pearl

ACE inhibitors ↓ efferent tone → ↓GFR → creatinine rise expected. Contraindicated in bilateral renal artery stenosis (precipitates acute renal failure) and pregnancy (teratogenic).

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Embryology

Kidney develops from intermediate mesoderm through 3 stages: Pronephros (week 4; regresses) → Mesonephros (weeks 4-8; male genital ducts) → Metanephros (permanent kidney).

  • Ureteric bud (from mesonephric duct) → ureter, pelvis, calyces, collecting ducts
  • Metanephric mesenchyme → glomerulus, PCT, loop of Henle, DCT
⭐ Boards Pearl

Horseshoe kidney = lower poles fuse; caught on IMA → stays at L3; most common renal fusion anomaly; associated with Turner syndrome. Potter sequence = bilateral renal agenesis → oligohydramnios → lung hypoplasia + limb deformities + facial anomalies.

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Clinical Pearls

ConditionKey FeaturesUrinalysis
Nephrotic syndrome>3.5 g/day protein; edema; hyperlipidemia; hypercoagulableFatty casts; oval fat bodies
Nephritic syndromeHematuria; HTN; oliguria; mild proteinuriaRBC casts (pathognomonic)
Minimal change diseaseMost common nephrotic in kids; podocyte effacementMassive proteinuria; no RBC
IgA nephropathyMost common GN worldwide; hematuria 1-3d after URIHematuria (gross)
CKDGFR <60 >3 months; anemia, hyperK, acidosis, renal osteodystrophyWaxy/broad casts
🩺 Clinical

Dialysis indications — AEIOU: Acidosis (refractory), Electrolytes (hyperkalemia), Intoxication, Overload (fluid refractory to diuretics), Uremia (pericarditis, encephalopathy).