Anatomy

Integumentary System

Skin layers, appendages, cells, and neurovascular supply — the body's largest organ

Skin Layers Overview

The skin (integument) has three main layers: epidermis, dermis, and hypodermis (subcutaneous tissue). It is the largest organ of the body (~1.5-2 m² surface area, 4-5 kg).

LayerTissue TypeKey Contents
EpidermisStratified squamous epitheliumKeratinocytes, melanocytes, Langerhans cells, Merkel cells; NO blood vessels
Dermis (papillary)Loose connective tissueDermal papillae, capillary loops, Meissner corpuscles (light touch)
Dermis (reticular)Dense irregular connective tissueCollagen I and III, elastin, hair follicles, glands, Pacinian corpuscles (pressure)
HypodermisAdipose and loose connective tissueFat storage, thermoregulation, Ruffini endings (stretch); NOT part of skin proper
⭐ Boards PearlEpidermis is avascular — oxygen and nutrients reach it by diffusion from dermal capillaries. This is why thin skin grafts (split-thickness) can survive initially without blood supply.

Epidermis — Layers (Deep to Superficial)

LayerLatin NameKey Features
Stratum basaleGerminativumDeepest; mitotically active; contains melanocytes and Merkel cells; attached to basement membrane
Stratum spinosumSpinosumPrickle cells; desmosomes (strong cell junctions); site of acantholysis in pemphigus vulgaris
Stratum granulosumGranulosumKeratohyalin granules; lamellar bodies release lipids (waterproof barrier)
Stratum lucidumLucidumOnly in thick skin (palms, soles); clear, dead cells
Stratum corneumCorneumOutermost; dead, anucleate, keratin-filled; barrier function; shed (desquamation) ~14 days
🧐 Mnemonic — Layers deep to surface"Come, Let's Get Sun-burned" = Basale, Spinosum, Granulosum, Lucidum, Corneum (or "Californians Like Girls in String bikinis")

Dermis and Hypodermis

The dermis provides mechanical strength via type I collagen (most abundant) and elasticity via elastin fibers. The dermis-epidermis junction has rete ridges (epidermis projecting down) and dermal papillae (dermis projecting up) — this undulating interface increases adhesion surface area.

  • Type I collagen — mature skin, tendons (most abundant collagen)
  • Type III collagen — fetal skin, wound healing, blood vessels (reticulin)
  • Type IV collagen — basement membrane
  • Elastin — provides recoil; degrades with UV exposure (solar elastosis)
🩹 Clinical NoteEhlers-Danlos syndrome: defect in collagen synthesis/processing → hyperextensible skin, joint hypermobility, easy bruising. Type depends on collagen affected.

Skin Cell Types

CellOriginLocationFunction / Relevance
KeratinocytesEctodermAll epidermal layers90% of epidermis; produce keratin; barrier function
MelanocytesNeural crestStratum basaleProduce melanin (UV protection); 1:10 ratio to keratinocytes; melanoma origin
Langerhans cellsBone marrow (monocyte)Stratum spinosumAntigen-presenting cells; destroyed by UV; express CD1a; Birbeck granules on EM
Merkel cellsNeural crestStratum basaleSlow-adapting mechanoreceptors (fine touch, sustained pressure); Merkel cell carcinoma
FibroblastsMesodermDermisCollagen and elastin synthesis; wound healing
Mast cellsBone marrowDermis (near vessels)IgE-mediated degranulation; histamine release; urticaria, anaphylaxis

Skin Appendages

AppendageOriginFunctionKey Points
Hair follicleEpidermis (downgrowth)Hair production, thermoregulationArrector pili (sympathetic adrenergic) = goosebumps; stem cells in bulge region
Eccrine sweat glandsEpidermisThermoregulation (aqueous sweat)Cholinergic innervation (sympathetic but ACh); all over body; most numerous on palms/soles
Apocrine sweat glandsEpidermisPheromones, emotional sweatAxilla, groin, areola; active after puberty; odor from bacterial breakdown
Sebaceous glandsEpidermisSebum (lipid-rich, waterproofing, antimicrobial)Attached to hair follicles; androgen-stimulated; acne = plugged pore + P. acnes
NailsEpidermisProtection, fine manipulationNail matrix (under lunula) = nail growth; ~3 mm/month fingernails
⭐ Boards PearlEccrine glands = cholinergic even though sympathetic. All other sympathetic effectors use norepinephrine. This is a classic pharmacology/autonomic boards question.

Skin Functions

  • Protection — physical barrier (keratin, lipids), UV protection (melanin), antimicrobial (acidic pH ~5.5, sebum, defensins)
  • Thermoregulation — vasodilation/constriction, eccrine sweating (evaporative cooling)
  • Sensation — mechanoreceptors (Meissner, Pacinian, Ruffini, Merkel), free nerve endings (pain, temperature)
  • Vitamin D synthesis — UV-B converts 7-dehydrocholesterol → previtamin D3 → vitamin D3 (cholecalciferol); converted to active 1,25-OH vitamin D in kidney
  • Immune function — Langerhans cells, mast cells, IgA in sweat
  • Fluid barrier — prevents transepidermal water loss (TEWL)

Clinical Pearls

⭐ Burn Classification1st degree (superficial): epidermis only, painful, erythema, no blisters (sunburn). 2nd degree (partial thickness): epidermis + part of dermis, very painful, blisters. 3rd degree (full thickness): all layers destroyed, painless (nerve destruction), leathery. 4th degree: to bone/muscle.
🩹 Wound Healing PhasesHemostasis (immediate): platelet plug, clot. Inflammation (days 1-4): neutrophils then macrophages; debridement. Proliferative (days 4-21): fibroblasts, collagen III, angiogenesis, granulation tissue. Remodeling (weeks-years): collagen III replaced by I; scar matures; tensile strength never exceeds 80% of original.
🧐 ABCDE of MelanomaAsymmetry, Border irregularity, Color variation, Diameter greater than 6mm, Evolution (change over time). Most common in males: back. Most common in females: legs.
⭐ Sensory Receptors Quick ReferenceMeissner corpuscles (papillary dermis) = light touch, two-point discrimination. Pacinian corpuscles (reticular dermis/hypodermis) = pressure, vibration. Ruffini endings (dermis) = skin stretch. Merkel discs (stratum basale) = sustained pressure, fine touch. Free nerve endings = pain, temperature, itch.